
The Ongoing Oceanic Catastrophe In Australia Was Predicted Years Ago
A sea lion with domoic acid poisoning experiencing involuntary muscle spasms.
We have another reason to address warming waters, extreme weather events and inefficient agriculture. Put together, they contribute to the increased risk of harmful algal blooms (HABs) that can bring mass death to the marine ecosystems they pervade. California can report 50 dead dolphins in a week, while 'feral, demonic sealions' are attacking surfers.
Harmful Algal blooms have been recorded for hundreds of years, with a link seen between the discolored lake water, and the subsequent death of the animals that drank from it. Over the last 40 years or so, these events have become more frequent. This beloved dog, Cedar, died within hours from a 'benthic algae mat'. These formations of algae grow at the bottom of a body of water and detach, surfacing and posing a distinct littoral threat. They're harder to detect than surface blooms and may be more persistent. They can enter the food web and threaten the rest of us via shellfish, while their 'toxic aerosol' can cause respiratory problems.
Public Safety Poster about the dangers of benthic algal mats.
Warmer Waters Favor Cyanobacteria
Blue-green algae isn't algae but in fact cyanobacteria, which are prokaryotes that photosynthesize. Different types of organisms have their environmental preferences and cyanobacteria is the same. It prefers warm waters compared to similar microorganisms so it will thrive thanks to global warming. Interestingly, the darkness of this top layer of cyanobacteria can absorb more heat, creating a feedback loop that perpetuates its growth.
Climate action strategies include recognizing tipping points and taking early action. This applies to HABs the same as it does to die-back in the Amazon.
The body of water on the right has a cyanobacteria bloom. The left has no bloom.
Cause and Effect of Bad Water Management
Nutrient run-off is the leading, controllable cause of algal blooms. The water wasted from flood irrigation gives us enough reason to adopt more efficient methods of irrigation, even before considering the risk of runoff. Rather than flooding a field and having some of that water overflow, and enter the local environment, other methods are more targeted. Unlike flooding, other techniques (for example drip irrigation) do not cause water to overflow the field and runoff into a tributary, where the nutrient pollution enters the wider aqueous ecosystem.
Nutrient pollution is a problem in more than one-third of lakes and half of all rivers and streams in the United States. While preventing the runoff upstream will be a way to prevent the blooms, the EPA has a list of local resources to deal with them.
Beyond creating toxins that can directly kill animals, or harm humans that eat contaminated animals, people may suffer skin irritation and respiratory problems from contact with some of these microorganisms.
Species of algae that aren't toxic can still cause harm by forming a surface layer that blocks sunlight for the plant life below. Bodies of water can deteriorate into 'dead zones,' where this lack of sunlight kills the aquatic plants below, now unable to photosynthesize, which depletes the water of oxygen. With that, limited fish or marine life can survive.
The largest dead zone in America occurs every summer, when nutrient pollution from the Mississippi River Basin enters the Gulf of America and creates a dead zone of about 4,300-6,500 square miles. Last year saw the dead zone reach 6,700 square miles; the EPA estimates that it will cost $7 Billion a year to reduce this problem.
Reasons to Like Algae
Having explored toxic algae and the hazards that can arise from benign algae, let's ponder what redeeming uses this group of organisms may offer. Some algae can be used as fertilizers, which would address the ammonia production carbon emissions issues. It's further beneficial as a form of carbon capture and sequestration: while the Haber Bosch would require intensive energy input which causes high emissions (1.8% of global CO2 emissions), algae can grow naturally in a carbon intensive environment, removing it and creating biomass. This biomass has applications beyond fertilizers, such as in biofuels. Spirulina adds a boost to your morning smoothie, and dried seaweed is delicious.
Sewage leaks are inherently disgusting and bad for drinking water, and lead to HABs that are the same. Sewage is nutrient pollution that fuels blooms, giving municipalities further reason to efficiently address sewage problems, if they needed it. Algae is not just caused by wastewater, but could help address the issue. From a research paper about using algae for biomass production and nutrient separation while treating wastewater: 'When algae were used to clean wastewater, amazing benefits were guaranteed, such as a decrease in the formation of dangerous solid sludge and the creation of valuable algal biomass through recycling of the nutrients in the wastewater.'
Let us work to limit the causes of harmful algae while harnessing the benefits of helpful algae.
The First Plague in the Book of Exodus (from The Bible) talks about the sea turning red, of fish dying, of people being unable to drink from the odorous waters. These sorts of events used to be rare, natural occurrences, but over the last 40 years these plagues have become more frequent. Australia is in the midst of an 'ecological catastrophe' as a HAB 70 times the size of Sydney Harbor disrupts life in South Australia, with residents protesting for action. Commercial fisherman are facing bankruptcy, while all residents are suffering.
Dead Maori Octopus
Could this have been prevented? Yes, a dozen local researchers wrote to the Federal Environment Minister in 2023 about the importance of mitigation and monitoring, and requested funding for reef protection. They were ignored.
The Marine Fisher Association says that the current Harmful Algal Bloom has caused 'complete ecosystem failure.'
Hashtags

Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles
Yahoo
32 minutes ago
- Yahoo
Ryan Gosling's Latest Appearance Is a 180 From His Wild Sci‑Fi Transformation
Ryan Gosling's Latest Appearance Is a 180 From His Wild Sci‑Fi Transformation originally appeared on Parade. Ryan Gosling is ditching his wild beard and long locks from the sci-fi transformation he debuted earlier this summer. Over the weekend, the 44-year-old actor stopped by San Diego Comic-Con where he appeared on a panel to discuss his upcoming film, Project Hail Mary. While Gosling was on hand to discuss his on screen journey to space, he looked nothing like his character. In the trailer, which premiered in June, Gosling was almost unrecognizable with a bushy beard and unwashed, stringy hair — and at Comic-Con, Gosling was back to his usual, well-kept self. He was joined by the film's directors Phil Lord and Christopher Miller as well as screenwriter Drew Goddard and author Andy Weir, who penned The Martian, the novel that inspired the film. 🎬 SIGN UP for Parade's Daily newsletter to get the latest pop culture news & celebrity interviews delivered right to your inbox 🎬 The movie is set to follow middle school teacher Ryland Grace who is tasked with traveling to space, 11.9 light-years away, in order to figure out why the sun is dying and save Earth from extinction. In the midst of his endeavor, Grace wakes up on a spaceship, confused about why he is there and lacking any recollection of his past. Although the teacher attempts to bow out of the massive responsibility, he quickly learns that he doesn't have a choice in the matter. While discussing his physical transformation for the role at the event, Gosling jokingly referred to his character as "a space caveman, in this placenta onesie" — noting that he hopes to see the placenta onesie at Comic-Con in the future. Gosling added that he and the writers "tried to create these different layers to him as he's evolving from a space caveman to a space person that needs to do some really important stuff." "What's so inspiring about him is he reacts to a lot of things like I might or a lot of us might, and he's terrified appropriately of the task at hand," Gosling said, per PEOPLE. "He's somebody who on Earth had given up on himself, and has been given this opportunity to believe in himself again." Project Hail Mary is set to premiere March 20, 2026. Watch the Project Hail Mary trailer below. Ryan Gosling's Latest Appearance Is a 180 From His Wild Sci‑Fi Transformation first appeared on Parade on Jul 27, 2025 This story was originally reported by Parade on Jul 27, 2025, where it first appeared. Solve the daily Crossword
Yahoo
2 hours ago
- Yahoo
Millions of people are suffering from brain fog. A new study will find out why
Millions of people who recover from infections like COVID-19, influenza and glandular fever are affected by long-lasting symptoms. These include chronic fatigue, brain fog, exercise intolerance, dizziness, muscle or joint pain and gut problems. And many of these symptoms worsen after exercise, a phenomenon known as post-exertional malaise. Medically the symptoms are known as myalgic encephalomyelitis or chronic fatigue syndrome (ME/CFS). The World Health Organization classifies this as a post viral fatigue syndrome, and it is recognised by both the WHO and the United States Centers for Disease Control and Prevention as a brain disorder. Experiencing illness long after contracting an infection is not new, as patients have reported these symptoms for decades. But COVID-19 has amplified the problem worldwide. Nearly half of people with ongoing post-COVID symptoms – a condition known as long-COVID – now meet the criteria for ME/CFS. Since the start of the pandemic in 2020, it is estimated that more than 400 million people have developed long-COVID. To date, no widely accepted and testable mechanism has fully explained the biological processes underlying long-COVID and ME/CFS. Our work offers a new perspective that may help close this gap. Our research group studies blood and the cardiovascular system in inflammatory diseases, as well as post-viral conditions. We focus on coagulation, inflammation and endothelial cells. Endothelial cells make up the inner layer of blood vessels and serve many important functions, like regulating blood clotting, blood vessel dilation and constriction, and inflammation. Our latest review aims to explain how ME/CFS and long-COVID start and progress, and how symptoms show up in the body and its systems. By pinpointing and explaining the underlying disease mechanisms, we can pave the way for better clinical tools to diagnose and treat people living with ME/CFS and long-COVID. What is endothelial senescence? In our review, our international team proposes that certain viruses drive endothelial cells into a half-alive, 'zombie-like' state called cellular senescence. Senescent endothelial cells stop dividing, but continue to release molecules that awaken and confuse the immune system. This prompts the blood to form clots and, at the same time, prevent clot breakdown, which could lead to the constriction of blood vessels and limited blood flow. By placing 'zombie' blood-vessel cells at the centre of these post-viral diseases, our hypothesis weaves together microclots, oxygen debt (the extra oxygen your body needs after strenuous exercise to restore balance), brain-fog, dizziness, gut leakiness (a digestive condition where the intestinal lining allows toxins into the bloodstream) and immune dysfunction into a single, testable narrative. From acute viral infection to 'zombie' vessels Viruses like SARS-CoV-2, Epstein–Barr virus, HHV-6, influenza A, and enteroviruses (a group of viruses that cause a number of infectious illnesses which are usually mild) can all infect endothelial cells. They enable a direct attack on the cells that line the inside of blood vessels. Some of these viruses have been shown to trigger endothelial senescence. Multiple studies show that SARS-CoV-2 (the virus which causes COVID-19 disease) has the ability to induce senescence in a variety of cell types, including endothelial cells. Viral proteins from SARS-CoV-2, for example, sabotage DNA-repair pathways and push the host cell towards a senescent state, while senescent cells in turn become even more susceptible to viral entry. This reciprocity helps explain why different pathogens can result in the same chronic illness. Influenza A, too, has shown the ability to drive endothelial cells into a senescent, zombie-like state. What we think is happening We propose that when blood-vessel cells turn into 'zombies', they pump out substances that make blood thicker and prone to forming tiny clots. These clots slow down circulation, so less oxygen reaches muscles and organs. This is one reason people feel drained. During exercise, the problem worsens. Instead of the vessels relaxing to allow adequate bloodflow, they tighten further. This means that muscles are starved of oxygen and patients experience a crash the day after exercise. In the brain, the same faulty cells let blood flow drop and leak, bringing on brain fog and dizziness. In the gut, they weaken the lining, allowing bits of bacteria to slip into the bloodstream and trigger more inflammation. Because blood vessels reach every corner of the body, even scattered patches of these 'zombie' cells found in the blood vessels can create the mix of symptoms seen in long-COVID and ME/CFS. Immune exhaustion locks in the damage Some parts of the immune system kill senescent cells. They are natural-killer cells, macrophages and complement proteins, which are immune molecules capable of tagging and killing pathogens. But long-COVID and ME/CFS frequently have impaired natural-killer cell function, sluggish macrophages and complement dysfunction. Senescent endothelial cells may also send out a chemical signal to repel immune attack. So the 'zombie cells' actively evade the immune system. This creates a self-sustaining loop of vascular and immune dysfunction, where senescent endothelial cells persist. In a healthy person with an optimally functioning immune system, these senescent endothelial cells will normally be cleared. But there is significant immune dysfunction in ME/CFS and long-COVID, and this may enable the 'zombie cells' to survive and the disease to progress. Where the research goes next There is a registered clinical trial in the US that is investigating senescence in long-COVID. Our consortium is testing new ways to spot signs of ageing in the cells that line our blood vessels. First, we expose healthy endothelial cells in the lab to blood from patients to see whether it pushes the cells into a senescent, or 'zombie,' state. At the same time, we are trialling non‑invasive imaging and fluorescent probes that could one day reveal these ageing cells inside the body. In selected cases, tissue biopsies may later confirm what the scans show. Together, these approaches aim to pinpoint how substances circulating in the blood drive cellular ageing and how that, in turn, fuels disease. Our aim is simple: find these ageing endothelial cells in real patients. Pinpointing them will inform the next round of clinical trials and open the door to therapies that target senescent cells directly, offering a route to healthier blood vessels and, ultimately, lighter disease loads. Burtram C. Fielding is Dean Faculty of Sciences and Professor in the Department of Microbiology, Stellenbosch University This article is republished from The Conversation under a Creative Commons license. Read the original article.


Gizmodo
2 hours ago
- Gizmodo
The Directors of ‘Project Hail Mary' Explain Why the Movie Is a PC, Not a Mac
Making movies is all about compromises. This actor is unavailable, so you cast someone else. That location is too expensive, so let's build a set. This shot is impossible, so let's think of something better. At every step, the big, huge mechanism of filmmaking is always a work in progress. But on Project Hail Mary, directors Chris Miller and Phil Lord tried to embrace a new philosophy. 'What's great about this movie is there are so many things that make it harder to make,' Miller said in Hall H at San Diego Comic-Con. 'All of the zero G, all of the centrifugal gravity, the characters have to have a wall between them because their atmospheres are different. Everything that a regular movie would be like, 'Oh, we can change that,' we were like, 'Anything that makes it harder we're not going to change.' We're going to stay true to it, and then that difficulty is what makes it interesting and makes it special.' His co-director, Phil Lord, put it another way. 'We kept saying, with respect, this movie is not a Mac, it's a PC,' he said, to much laughter. 'The movie is a machine, the ship is a machine; it can be beautiful, it just can't be pretty.' It is true that almost everything about Project Hail Mary makes it seem incredibly difficult to make. Most of the movie is set on a spaceship. That spaceship meets an alien race represented by a creature made out of stone that doesn't speak English. Changing the setting or the character could've still conveyed the overall idea of the story, but it wouldn't be the story author Andy Weir wrote in his novel. So everything had to be right. So how did Lord and Miller bring that rock creature, nicknamed Rocky, to life? 'We called our friend Neil Scanlan at the Lucasfilm creature shop, and we tackled it together,' Miller said. 'We built a practical creature that was puppeteered by an amazing puppeteer named James Ortiz and a team of five, which we called the Rocketeers, and it was amazing having Rocky there on set every day so that we could have a real interaction and shoot the whole thing practically. Ultimately, it's going to end up being a beautiful blend of creature puppetry and animation, and he comes alive in a way that you would die for this character.' Sounds like it's beautiful, but maybe not pretty, just like a PC. Project Hail Mary, starring Ryan Gosling, opens in theaters on March 20. Want more io9 news? Check out when to expect the latest Marvel, Star Wars, and Star Trek releases, what's next for the DC Universe on film and TV, and everything you need to know about the future of Doctor Who.