Walmart Recall Update: Customers Told Not to Consume Products Nationwide
Newsweek AI is in beta. Translations may contain inaccuracies—please refer to the original content.
Walmart has so far announced that four food products sold in its stores nationwide have been recalled in July.
The latest, on Monday, was Danone U.S.' voluntarily recall of all flavors and sizes of its YoCrunch yogurt, following consumer complaints about the potential presence of sharp, transparent plastic pieces in the yogurt's dome topper, the company said.
Why It Matters
This recall marks the latest in a series of notable product recalls issued so far this year.
Food recalls due to contamination or mislabeling can undermine consumer trust and place vulnerable individuals (such as children, the elderly, or those with allergies) at greater risk.
Food safety remains a paramount concern for American households, and the frequency of such issues may influence shopping habits and regulatory responses nationwide.
A Walmart Supercenter cart sits outside of the store on February 20, 2024, in Hallandale Beach, Florida.
A Walmart Supercenter cart sits outside of the store on February 20, 2024, in Hallandale Beach, Florida.Full List of Walmart Product Recalls in July 2025
July 2 — Oscar Mayer Turkey Bacon (Kraft Heinz)
Kraft Heinz pulled nearly 368,000 pounds of fully-cooked Oscar Mayer Turkey Bacon after in-house tests suggested possible Listeria monocytogenescontamination. The bacon—sold nationwide, including at Walmart—carries "use by" dates from July 18 to Sept 4 and lot codes RS19, RS40 or RS42. No illnesses have been reported, but shoppers are urged to throw out or return the product.
July 8 — RITZ Peanut Butter Cracker Sandwiches (Mondelēz)
Mondelēz voluntarily recalled select 8-, 20- and 40-count cartons of RITZ Peanut Butter Cracker Sandwiches—and a variety pack—because some individual packs were mis-wrapped in "Cheese" film, hiding the peanut allergen inside. The mislabeled snacks, sold nationwide at retailers such as Walmart, pose a serious risk to people with peanut allergies. No injuries have been reported.
July 10 — Lewis Bake Shop Artisan Style Half-Loaf (Hartford Bakery)
Hartford Bakery yanked six lots of its Lewis Bake Shop Artisan Style Half-Loaf bread after visible hazelnuts turned up in packaging that failed to disclose the tree-nut ingredient. About 883 loaves reached Walmart and other stores across 12 states. One minor digestive complaint has surfaced; customers can return the bread for a full refund.
July 14 — YoCrunch Yogurts (Danone U.S.)
Danone recalled all flavors and sizes of its YoCrunch yogurt line after consumers found clear plastic shards—7 to 25 mm long—in the dome toppers. The nationwide recall, affecting multipacks and single 6-oz cups sold at Walmart and other chains, was issued over choking concerns. Consumers should discard the yogurts or seek a refund through YoCrunch's helpline.
What People Are Saying
Walmart says in a statement on its product recall webpage: "Walmart and Sam's Club are committed to the health and safety of our customers and members and to providing products that are safe and compliant, all supported by our health and wellness, product safety, and food safety professionals. In the event of a product recall, we work swiftly to block the item from being sold and remove it from our stores and clubs."
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Postpartum Thyroiditis: Risk Factors, Workup, and Management
This transcript has been edited for clarity. Kaniksha Desai, MD: Welcome to the Thyroid Stimulating Podcast. This podcast was created in partnership with the American Thyroid Association to discuss up-to-date diagnosis and management of a wide array of thyroid diseases. I'm your host, Dr Kaniksha Desai, and today we're talking about a condition that's frequently underrecognized in clinical practice: postpartum thyroiditis. This autoimmune thyroid disorder affects up to 5%-10% of women in the first year after giving birth. It typically starts with the phase of hyperthyroidism, which may be mistaken for the normal demands of the postpartum period. This is then followed by hypothyroidism, which can significantly impact energy, mood, and recovery. While most women eventually return to normal thyroid function, some develop permanent hypothyroidism. In today's episode, we'll cover the typical timeline and phases of postpartum thyroiditis; risk factors and diagnostic workup; management strategies, including when to treat and when to monitor; and how to counsel patients navigating this condition. Joining us today is Dr Caroline Nguyen, clinical associate professor at the Keck School of Medicine of USC. Her clinical research expertise is in the diagnosis and management of thyroid disorders during pregnancy. Her work spans hyperthyroidism, hypothyroidism, and thyroid cancer, as well as postpartum thyroiditis. She is published in leading journals, including Thyroid and JCNM, and Dr Nguyen is also a member of the writing group for the upcoming thyroid and pregnancy guidelines expected later this year. Thank you for joining us today. We're excited to have you talk about postpartum thyroiditis. Caroline T. Nguyen, MD: Thank you, Dr Desai, for having me. Desai: For our listeners, can you briefly define postpartum thyroiditis? Nguyen: Originally, it was described as the occurrence of de novo, transient thyroid dysfunction in the first year after pregnancy in a previously euthyroid person. However, this definition is a bit limited and now we think of it slightly differently. We think of it as a pattern of transient, autoimmune thyroid dysfunction that occurs within the 12 months after delivery. Essentially, it's a form of silent thyroiditis — silent in that it's not typically associated with pain. It involves a form of chronic lymphocytic infiltration and tissue injury, leading to a release of large amounts of thyroid hormone into the circulation, as you mentioned. What's unique about it is that it occurs in the postpartum period. Why may that be? The hypothesis is that during pregnancy, there's suppression of the immune system, and that's most prominent in the third trimester. This is why many autoimmune conditions improve in pregnancy, such as Graves disease. Often, patients can come off of their antithyroid drugs during the third trimester. What happens is that, as pregnancy progresses, the immune system is suppressed and then the immune system rebounds postpartum. This is when autoimmune conditions may present. Interestingly, we see postpartum thyroiditis occur earlier in the postpartum period. We think it's because it's associated with cellular immunity, while conditions like Graves disease tend to occur later in the postpartum period, and the hypothesis is that it is the association with humoral immunity and antibody formation. Desai: That helps us. I just want to clarify that this is different from the Graves recurrence. When do you suspect this vs Graves disease or Hashimoto's? Nguyen: Typically, you can tell by timeline in the postpartum period. Generally, postpartum thyroiditis tends to happen a little earlier. Occurrence tends to happen where usually, within 1-6 months, you should have presentation of symptoms, whereas Graves tends to be in the latter part of the postpartum period, maybe after 6-12 months. Those are just general guidelines and there's definitely going to be overlap in the middle. You bring up a really good point that it can be challenging to tell the difference between the two because they both present with hyperthyroid symptoms and hyperthyroid labs. A few things can help. Sometimes the clinical exam, like in Graves disease — if you have eye disease or if you have a goiter, then that would be more suggestive of Graves disease. You could also do laboratory testing if clinically you couldn't tell. Laboratory testing would be the thyroid function tests, so TSH, T4, plus or minus T3 could help. Between the two, what would help you the most to distinguish — because you could have overt hyperthyroidism in both — would be the antibodies. Usually the TRAb, or the TSH receptor binding antibodies, or thyroid-stimulating immunoglobulins, TSIs. Those are the antibodies that are responsible for Graves disease and the pathophysiology of Graves disease. If you have those present, then you should suspect that this is Graves disease more so than just postpartum thyroiditis. Desai: Is there any imaging that might help, like a thyroid ultrasound? Nguyen: We don't routinely order thyroid ultrasounds in the evaluation of this. Usually, you would have your clinical suspicion and then you would do your biochemical testing. The ultrasound, if you happen to have it already, could be helpful in the sense that typically with thyroiditis, you would see more of a heterogeneous architecture on the thyroid, maybe areas of hypoechogenicity. The thyroid gland itself could be normal in size or maybe slightly enlarged. The vascularity might be the most helpful in the sense that usually it would be reduced in thyroiditis in contrast to Graves disease, which would show increased vascularity. If you have an ultrasound in your clinic or already had one coming in, that could be helpful, but I would say the antibody testing tends to be easier. Desai: In nonpregnant women, we also use a thyroid uptake and scan. Can you talk a little bit about why that would or wouldn't work here? Nguyen: Typically, post-partum, we tend to try to avoid things like that because most of the time, patients might be breastfeeding. It's also a bit of a cumbersome study and it takes time and also isn't really necessary in the sense that we have so many other options that are so much easier. I think that's why it's has fallen from use. Desai: That's how we work it up. It's a biochemical-plus-clinical evaluation. We briefly talked about how it starts with hyperthyroidism, goes through hypothyroidism, and then comes out the other end normal in many people. Can you walk us through what a typical clinical course looks like? Nguyen: Patients often follow up in postpartum. It's common to have a 6-week follow-up with their OBs, and they might present with symptoms. In the hyperthyroid phase, it can present with symptoms like palpitations, maybe irritability, increased anxiety, heat intolerance, maybe a little bit of a tremor, or they might notice differences in their ability to breastfeed, and milk production can be altered. Those symptoms can sometimes bring patients in initially; however, it's important to note that some patients might have those symptoms and never notice them. They may just exit out of the hyper phase and then just present in the hypo phase. In the hypo phase, the symptoms are completely the opposite. You're going to have more of this fatigue, maybe cold intolerance, constipation, difficulty concentrating, impaired memory, dry skin — the classic symptoms of hypothyroidism. The symptoms are challenging because they can vary significantly depending on when the patient presents. Also, what makes it challenging post-partum is that many of these symptoms overlap with just being in the postpartum period and having sleep deprivation and a new baby to care for. It can be really hard just to base this on symptoms alone, but I do think it's really important for the clinician to ask patients about these symptoms. What happens is that many patients will just attribute any symptoms they develop in the postpartum period to being post-partum. They just think it's normal. I think clinicians need to make a better point of asking and seeing if there is more to it than just being post-partum. Desai: Would there be any benefit to just testing, like just getting that TSH test? Nguyen: Screening everybody. Desai: It's kind of hard to see which of these symptoms are postpartum vs which of them are truly symptomatic, unless it's an extreme, right? Nguyen: That raises a bigger question, this idea of universal thyroid screening. You bring up a good point because the estimated prevalence of postpartum thyroiditis, like you said, is 5%-10%. That's 1 in 12 patients who are going to have thyroid dysfunction. That's only estimated based on observational studies on clinically apparent disease, meaning that we're probably missing many patients who never present because they never talk about their symptoms or mention them. That's a bigger topic, about whether we should really be screening everyone, but definitely I think asking questions and screening for those symptoms, and then testing patients who are reporting these symptoms, makes a lot of sense. I want to bring up one thing about the testing that can sometimes be confusing. We test TSH often, and many people will order a TSH with reflex free T4. However, in the postpartum period, it can be a bit tricky. Maybe you could still order it and you could get it correctly, but it is important to look at both the TSH and the T4. If your lab doesn't — some labs, interestingly, don't reflex if the TSH is low. They only reflex if the TSH is high. In this scenario, you may want to order both because in that transition phase from hyper to hypo, you could have a THS that lags behind. That will look hyper but be low. When you get the T4 levels, they're low because they've already transitioned into the hypo phase. The TSH is lagging behind and hasn't caught up. I think in this scenario, looking at both can be helpful to know exactly where they are in the course of the postpartum. Desai: To clarify, initially, the free T4 goes up and then the TSH comes down, right? Then the free T4 will come down and then the TSH will go up? Then both of them will hopefully be normal? Nguyen: Correct. When you talk about the overall prognosis, generally the thought is that it'll last about 1 year, which is a long time, right? These patients do need to be followed because they are going to go through these transitions. When you start with hyper, and monitoring for hyper and treating conservatively for hyper, but then they switch to hypo, they should be offered treatment in the hypothyroid phase if they're symptomatic because it can be quite significant. Most of the time, these TSH levels can be double digits and really high because these patients are young. Offering treatment during that time would be important for symptom relief. You bring up a good point that, at 12 months, this process tends to resolve. It would be important at the 12-month period to assess for whether patients need to continue on medication or if they can be titrated off the medication. Many will be able to come off the medication at 12 months, but there is a portion of patients — the prevalence is anywhere from 5% to 20% — who may end up having to stay on thyroid hormone medication even after that 1 year post-partum. Desai: That's a large amount of information, so I want to break it down into little pieces. We talked about the hypothyroidism phase, but if we could go back to the hyperthyroidism in the beginning, is there anything we need to do for treatment for that phase? Nguyen: You can offer patients beta-blockers to help with the symptoms, just like we would in any kind of hyperthyroid condition. The beta-blockers can help significantly with the palpitations, the tremors, and the heat intolerance. Usually in postpartum, we prefer propranolol or metoprolol. Propranolol is nice. It's dosed about every 8 hours, so patients can take it almost on an as-needed basis. I find that many patients like that because sometimes the symptoms are more prominent at certain times of the day. If not, you can take metoprolol, which is longer-acting. We tend to avoid atenolol just because there seems to be increased transfer into breast milk. It's felt to be safe to use beta-blockers for short periods of time in the lowest effective dose in the postpartum period, and it can really help with symptoms. Even during lactation, it's acceptable. Usually we use it only for a few weeks, which I find is what most patients need. Desai: To clarify, the symptoms that it's controlling are the heart racing and what other symptoms? Nguyen: Maybe if they have a little bit of a tremor, the palpitations, and increasing feelings of anxiety; those tend to be what people commonly complain about. They can also present with heat intolerance. If you use a nonselective beta-blocker such as propranolol, it can help with all of those. Desai: Will this expedite the hyperthyroidism phase or it does nothing to the hyperthyroidism? Nguyen: No, it's just symptom control. Some patients who feel like the symptoms are mild but tolerable don't always opt to use beta-blockers. It's just for the patient who's really having a hard time. I think with the symptoms, like I said, many patients don't even notice in that early postpartum period because they have so much else going on. It can be helpful for certain patients. I think the treatment for hypo is a little more significant. Desai: To clarify, antithyroid drugs are not indicated here. Nguyen: That is correct. Because of the pathophysiology of the condition, antithyroid drugs wouldn't help. The issue is not about the overproduction of thyroid hormone; it's the release of preformed thyroid hormone already. We definitely wouldn't want to treat with antithyroid drugs, especially in an exposed mom and baby, too, if it's not necessary. Desai: For the hypothyroid phase, we talked about starting on thyroid hormone — and we're talking about levothyroxine. You briefly touched on the topic, but is there a TSH that you think people should be started on or it's just symptom control? Nguyen: I think it's symptoms, but also maybe biochemical, too. Even though outside of pregnancy, we will start patients with TSH levels greater than 10. We tend to recommend treatment for that. I think in this scenario, it could be the same, if not even a lower threshold to start treatment. If patients are already symptomatic, their TSH is elevated, and you know they're going through this process, it would make sense to give them some hormone support. Usually, it will present as overt hypothyroidism. Then we typically will say you can replace with full-dose replacement for overt hypothyroidism — overt meaning both the TSH and the T4 are going to be abnormal. That's 1.6 µg/kg/d, as we would in a nonpregnant patient. Desai: For mild, subclinical hypothyroidism, you would consider 0.8 µg, and then for overt, the full dose? Nguyen: If it's mild, you could definitely start on a lower dose and then watch and monitor the patient to see if it needs to be titrated up. Desai: This brings me to my next question: How frequently should you order labs? Nguyen: Typically, in the beginning when you first see the patient and you've made the diagnosis and it's hyper, it's going to be a transient process and it's going to resolve. Nothing about your treatment is going to be adjusted necessarily by the lab. In that part, I don't think you'd have to test too frequently but perhaps within 4-6 weeks later, because you are looking for when they begin to transition into the hypothyroid phase. That's a little bit tougher to predict, depending on when they present to you. That's what you're watching out for. When that starts to happen, again, you would tell them to stop the beta-blocker because they don't need it anymore at that point, then consider adding on the thyroid hormone if they would like to start treatment. If they're not, and they want to monitor and wait — because sometimes it takes a while for that T4 to become low — they can keep monitoring maybe every 4-6 weeks. Eventually, once you're in the hypothyroid phase and you've decided to treat them, then you're going to monitor every 6 weeks as you would in a nonpregnant patient that you're treating with thyroid hormone until you can optimize the thyroid hormone levels with the right dose. Desai: When do you recommend titrating off the thyroid hormone? You said the majority of them recover, right? Nguyen: Yes. At 12 months is when I start to think about bringing down the dose. If they're doing well at that point, I start to titrate down on the dose gradually, and again, repeating labs every 6 weeks to see how they tolerate the titration. Desai: For the people who are permanently hypothyroid, when you titrate down, their TSH increases and so you have to leave them on levothyroxine? Nguyen: Yes. Desai: For everyone who recovers, does this ever happen again with future pregnancies? Nguyen: Yes. That's actually one of the biggest risk factors. If somebody has had this happen in one pregnancy, there's a risk of about 70% that they are going to have it in a future pregnancy. That's really important to tell the patient and prepare them. It's also important that, in most of the patients who have postpartum thyroiditis, the other big risk factor is thyroid autoimmunity. If you already know you have thyroid peroxidase antibody positivity — less so with thyroglobulin antibody positivity, or findings on ultrasound that point toward thyroid autoimmunity — these are the risk factors for potentially having postpartum thyroiditis and also for having hypothyroidism. This can be helpful information as well for predicting who might be at risk going into a future pregnancy and who might be at risk for permanent hypothyroidism. Even those who recover and become euthyroid at 1 year, potentially 10%-50% of them, long term, may still develop permanent hypothyroidism. We have to keep watching these patients and also prepare them for future pregnancy because sometimes patients become hypothyroid in that phase and they are unaware of it, and then they go into a next pregnancy and could be hypothyroid pre-conception. They should check their thyroid function before the next pregnancy in the event that they did have more permanent hypothyroidism as a result. Desai: Are there any other risk factors, like age, family history, or other medical conditions, that we should be concerned about? Nguyen: Probably the best data are for other autoimmune conditions in the person themselves or a family history of autoimmune conditions. Those would be the other risk factors I would consider. Desai: This would include type 1 diabetes, lupus, and things like that. Nguyen: Yes. Desai: Should all of these patients see endocrinology? I know we're kind of limited in the number of endocrinologists out there, but how do you feel about them seeing endocrinology? When do they need to see endocrinology? Nguyen: Probably most of these patients never see endocrinology. These are patients who didn't know they had thyroid disease or a thyroid condition. More likely, they're going to be followed up by their OBs at the postpartum visits, or maybe by their primary clinician. I think everybody who cares for patients in pregnancy or around pregnancy needs to be aware of this diagnosis and ideally become comfortable with diagnosing it and treating it. I think patients that should see endocrinology are those where the clinician's not sure about the diagnosis. If they're not sure if it's postpartum thyroiditis, Graves disease, or maybe hyperthyroidism from some other etiology, then they should be referred to endocrinology. If the clinician just doesn't feel comfortable managing postpartum thyroiditis, they should refer to endocrinology. These patients do need to be followed for that entire year, where a large amount of change can happen during that year and then even after that year, when you are titrating down the dose. If OBs or primary clinicians became more familiar and comfortable, they could manage this, and probably with its high prevalence, it would be necessary to treat all the patients out there. Desai: Thank you so much. For all of the non-endocrinologists managing this, what are your three takeaways? Nguyen: Again, the first would be to really be aware of the high prevalence of postpartum thyroiditis, and also of the increase in thyroid dysfunction that can happen post-partum. Ask patients about it because they many not volunteer the information or even be aware of it. I think asking about it is step 1. Then if you make the diagnosis, offering symptomatic treatment is step 2. There's this thought that this is a transient process and it will resolve on its own, that it doesn't need to be treated. I think, for a patient to have abnormal thyroid function for a year during a time that is extremely stressful already and hard, such as the postpartum year, is a disservice to patients. They need to be informed that there are treatment options to help them feel better, not that this is just going to resolve on its own and they need to just get through it. That's Step two. Step 3: Counseling and contraception until a patient is euthyroid would be very important. Then, planning future pregnancies for the reasons we discussed, and monitoring these patients for long-term hypothyroidism. Desai: Thank you for joining us today and talking about this condition that we often overlook. It's a pretty important condition occurring at a very vulnerable time for many people. Thank you for joining us today, Dr Nguyen. Please stay tuned for next month's episode of the Thyroid Stimulating Podcast .
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