
Scientists discovery why one of the world's continents apart at double speed
The process stems from the East African Rift System (EARS), which is a 2,000-mile-long rift that began forming at least 22 million years ago and runs through the region where Africa's Great Lakes are located.
This rift marks the boundary between two tectonic plates: the Somali Plate and the Nubian Plate (part of the African Plate), which are gradually pulling away from each other.
Scientists have now identified a massive upwelling of hot, partially molten rock beneath the region, known as the African Superplume, which is driving this divergence.
Beneath the surface, intense heat and pressure from the superplume are weakening and cracking the Earth's outer layer, known as the lithosphere.
GPS measurements indicate that the plates are moving apart at a rate of about 0.2 inches per year, roughly the speed at which human fingernails grow.
Over time, this rifting could form a new ocean, potentially splitting off parts of Somalia, eastern Ethiopia, Kenya, and Tanzania to form a new landmass.
While the full separation was previously thought to take tens of millions of years, recent models suggest it could happen in one to five million years.
In the new study, scientists from the University of Glasgow in Scotland used data from Kenya's Menengai geothermal field to trace the isotopes of the noble gas neon.
This helped the team determine whether the forces splitting Africa apart originate deep within the Earth's mantle or are due to shallower surface tectonic processes.
They found that the gas likely comes from deep within the Earth, between the outer core and the mantle.
Lead author Professor Fin Stuart said in a statement: 'We have long been interested in how the deep Earth rises to surface, how much is transported, and just what role it plays on forming the large-scale topography of the Earth's surface.'
'Our research suggests that a giant hot blob of rock from the core-mantle boundary is present beneath East Africa, it is driving the plates apart and propping up the Africa continent so it hundreds of meters higher than normal,' Stuart added.
Using high-precision mass spectrometry, the team also identified a consistent chemical 'fingerprint' across a wide area.
This supports the theory that the EARS is fueled by a 'superplume,' rather than several smaller sources.
The study provides crucial insights into continental breakup and ocean formation, enabling researchers to comprehend similar processes that have shaped Earth's surface throughout history.
EARS spans from Ethiopia to Malawi, and massive cracks have appeared in recent years.
In 2005, a series of over 400 earthquakes in Ethiopia's Afar region led to the sudden appearance of a 37-mile long crack, providing an example of how dynamic forces works.
Similarly in 2018, a massive crack emerged in Kenya's Great Rift Valley, disrupting transportation and highlighting the ongoing nature of the continental split.
As the rift continues to widen, scientists predict that seawater from the Red Sea and the Indian Ocean will eventually flood the low-lying areas, creating a new ocean basin.
Ken Macdonald, a marine geophysicist, said: 'The Gulf of Aden and the Red Sea will flood over the Afar region and into the East African Rift Valley, giving rise to a new ocean.'
'Consequently, this part of East Africa will evolve into its own distinct continent,' he added.
Countries like Somalia, Kenya, and Tanzania could become part of a new landmass, effectively forming a separate continent.
While landlocked nations such as Uganda and Zambia might gain coastlines, altering trade routes and geopolitical dynamic.
This ongoing rifting leads to frequent earthquakes, volcanic eruptions, and large fractures across the landscape.
Hashtags

Try Our AI Features
Explore what Daily8 AI can do for you:
Comments
No comments yet...
Related Articles


The Independent
4 minutes ago
- The Independent
Study reveals the foods which help avoid illness in later life
What you eat could determine how many chronic illnesses you get later in life, scientists warn. Research has revealed that a healthy diet - such as the Mediterranean diet which is high in plants, fish and unsaturated fats - could slow down the accumulation of chronic diseases including dementia in older adults. Inflammatory diets full of processed meat and sugar may accelerate it. Researchers from Karolinska Institutet in Sweden studied four diets to investigate their impact on chronic diseases in older adults. Three of the diets studied were healthy and focused on the intake of vegetables, fruit, whole grains, nuts, legumes, unsaturated fats and reduced intake of sweets, red meat, processed meat and butter/margarine. The fourth diet, however, was pro-inflammatory and focused on red and processed meat, refined grains and sweetened beverages, with lower intake of vegetables, tea and coffee. Researchers followed the diets of 2,400 adults aged 60 and older in Sweden for 15 years and tracked their chronic conditions. Dietary intake was measured using food frequency questionnaires, and adherence to four dietary patterns: the Empirical Dietary Inflammatory Index (EDII), AHEI, the Alternate Mediterranean Diet (AMED), and the MIND (Mediterranean–DASH Intervention for Neurodegenerative Delay). Multimorbidity was defined as the number of chronic diseases and grouped by organ system - musculoskeletal, cardiovascular, and neuropsychiatric. The results published in the journal Nature Aging revealed those who followed the healthy diets had a slower development of chronic diseases. For example, long-term adherence to healthy dietary patterns, particularly the AMED, AHEI, and MIND, was linked to a slower accumulation of chronic diseases in older adults. This applied to cardiovascular disease and dementia, but not to diseases related to muscles and bones. But those who followed the pro-inflammatory diet, on the other hand, increased their risk of chronic diseases. 'Our results show how important diet is in influencing the development of multimorbidity in ageing populations,' said co-first author Adrián Carballo-Casla, postdoctoral researcher at the Aging Research Centre, Department of Neurobiology, Care Sciences and Society, Karolinska Institutet. The protective effects of diet may be explained by reduced inflammation, a key factor in aging-related diseases. Study authors want to further their research by identify the dietary recommendations that may have the greatest impact on longevity and the groups of older adults who may benefit most from them, based on their age, gender, psychosocial background and chronic diseases.


The Independent
9 hours ago
- The Independent
New study reveals which diets will help avoid illness in later life
What you eat could determine how many chronic illnesses you get later in life, scientists warn. Research has revealed that a healthy diet - such as the Mediterranean diet which is high in plants, fish and unsaturated fats - could slow down the accumulation of chronic diseases including dementia in older adults. Inflammatory diets full of processed meat and sugar may accelerate it. Researchers from Karolinska Institutet in Sweden studied four diets to investigate their impact on chronic diseases in older adults. Three of the diets studied were healthy and focused on the intake of vegetables, fruit, whole grains, nuts, legumes, unsaturated fats and reduced intake of sweets, red meat, processed meat and butter/margarine. The fourth diet, however, was pro-inflammatory and focused on red and processed meat, refined grains and sweetened beverages, with lower intake of vegetables, tea and coffee. Researchers followed the diets of 2,400 adults aged 60 and older in Sweden for 15 years and tracked their chronic conditions. Dietary intake was measured using food frequency questionnaires, and adherence to four dietary patterns: the Empirical Dietary Inflammatory Index (EDII), AHEI, the Alternate Mediterranean Diet (AMED), and the MIND (Mediterranean–DASH Intervention for Neurodegenerative Delay). Multimorbidity was defined as the number of chronic diseases and grouped by organ system - musculoskeletal, cardiovascular, and neuropsychiatric. The results published in the journal Nature Aging revealed those who followed the healthy diets had a slower development of chronic diseases. For example, long-term adherence to healthy dietary patterns, particularly the AMED, AHEI, and MIND, was linked to a slower accumulation of chronic diseases in older adults. This applied to cardiovascular disease and dementia, but not to diseases related to muscles and bones. But those who followed the pro-inflammatory diet, on the other hand, increased their risk of chronic diseases. 'Our results show how important diet is in influencing the development of multimorbidity in ageing populations,' said co-first author Adrián Carballo-Casla, postdoctoral researcher at the Aging Research Centre, Department of Neurobiology, Care Sciences and Society, Karolinska Institutet. The protective effects of diet may be explained by reduced inflammation, a key factor in aging-related diseases. Study authors want to further their research by identify the dietary recommendations that may have the greatest impact on longevity and the groups of older adults who may benefit most from them, based on their age, gender, psychosocial background and chronic diseases.


Daily Mail
11 hours ago
- Daily Mail
Two common viruses may 'reignite' cancer cells in people decades after remission
Cancer survivors could see the disease return with a vengeance if they contract a common virus, new research suggests. A study, led by scientists from the University of Colorado Anschutz Medical Campus, looked at how Covid-19 and influenza could awaken dormant breast cancer cells that have spread to the lungs. In remission, dormant cancer cells can exist undetected in the body and may reactivate years later, potentially leading to the disease's spread. During the Covid-19 pandemic, anecdotal reports suggested a possible increase in cancer death rates, bolstering the idea that severe inflammation from the virus might play a role in arousing dormant cancer cells. Researchers tested this hypothesis in a lab setting using mice with breast cancer tumors and dormant cancer cells in their lungs. The mice were exposed to either SARS-CoV-2 (the virus that causes Covid-19) or the influenza, flu, virus. In both cases, the respiratory infections triggered the reawakening of dormant cancer cells in the lungs. Within days of infection the cancer cells rapidly spread in the mice and the secondary tumors appeared within two weeks. Commenting on the findings, study supervisor Dr James Degregori said: 'Dormant cancer cells are like the embers left in an abandoned campfire, and respiratory viruses are like a strong wind that reignites the flames.' A further analysis found that the awakening of dormant cancer cells is driven by the release of an inflammatory immune protein, called interleukin-6 (IL-6). In an effort to fight off viruses, the body ramps up production of various immune cells, including IL-6. But these well-intentioned molecules can lead to out-of-control inflammation in other areas of the body, which in turn, reawakens cancer cells in remission. The researchers say that the identification of IL-6 as a key trigger could help in developing new treatment plans to stop the chain reaction from happening. Dr Aguirre-Ghiso suggests the possibility of using using IL-6 inhibitors or other targeted immunotherapies to prevent the cancer from returning and spreading. Along with the lab study, the researchers analyzed two large health databases and found support for their hypothesis that respiratory infections can recharge cancer among patients who are in remission. The UK Biobank has information on more than 500,000 participants who were diagnosed with cancer and other diseases prior to the Covid-19 pandemic. Researchers from Utrecht University and Imperial College London, who collaborated with the University of Colorado Anschutz Medical Campus team, investigated whether a Covid-19 infection increased the risk of death among cancer patients. They focused on cancer survivors who had been diagnosed at least five years before the pandemic, ensuring they were likely in remission. This took into account all cancers in different parts of the body, not just in the breast. Among them, 487 individuals tested positive for Covid-19 and these were compared to 4,350 matched controls who tested negative. After excluding those cancer patients who died from Covid-19, the researchers found that cancer patients who tested positive for the virus faced nearly double risk of dying from the disease compared to those patients with cancer who had tested negative. 'The effect was most pronounced in the first year after infection,' said Dr Roel Vermeulen of Utrecht University. The researchers said the rapid progression of cancer they found when analyzing the data banks mirrored the quick expansion of dormant cancer cells in the mouse study. From the second population study, Dr Junxiao Hu and Dr Dexiang Gao drew data from the US Flatiron Health database pertaining to female breast cancer patients seen at 280 cancer clinics in the US. In the US, there are more than 300,000 new cases of breast cancer and over 42,000 deaths annually. These numbers include both invasive and non-invasive cases for women, as well as cases in men. They compared the incidence of tumors spreading to the lung among Covid-19-negative patients and those who tested positive for the virus (36,216 and 532 patients respectively). During the follow-up period of approximately 52 months, those patients who came down with Covid-19 were almost 50 percent more likely to experience metastatic progression to the lungs compared with patients with breast cancer who did not contract the disease. In conclusion, Dr Vermeulen said: 'Our findings suggest that cancer survivors may be at increased risk of metastatic relapse after common respiratory viral infections. 'It is important to note that our study focused on the period before COVID-19 vaccines were available.' The researchers SAID their findings, published in the scientific journal Nature, indicate that individuals with a history of cancer 'may benefit from taking precautions against respiratory viruses, such as vaccination when available, and discussing any concerns with their healthcare providers'.