logo
5 Symptoms Of Salmonella As Tomatoes Linked To Dozens Of Cases

5 Symptoms Of Salmonella As Tomatoes Linked To Dozens Of Cases

Yahooa day ago
Food safety watchdogs have linked 2024′s unusually high incidence of salmonella to tomatoes, The Independent shared.
UKHSA data showed that the UK saw a salmonella peak last year, with 17.1% rise compared to 2023 (the highest figures of the decade).
The Salmonella Strathcona outbreak, which is particularly dangerous, and the Salmonella Blockley strain, which is concerning because it's not previously been seen much in Europe, were both linked to tomatoes.
'Tomatoes were... blamed for a Salmonella Strathcona outbreak with 24 patients,' Food Safety News shared, while 84 Salmonella Blockley cases were said to come from the fruit.
It's also been associated with eggs and red meat.
Early 2025 data shows salmonella reports were high in the first quarter of this year, too.
Which tomatoes have been linked to salmonella in the UK?
We don't know which tomatoes in particular were linked to the disease, or where they came from.
We also don't know where they were sold in the UK.
But usually, salmonella in tomatoes comes from water supplies as they grow.
Worker hygiene and nearby animals can also affect susceptibility, CBS News added.
Can you wash salmonella off tomatoes?
You should always wash your produce to prevent disease.
But Virginia Tech food microbiologist Robert Williams told CBS News: 'We know you can wash off some salmonella... [but] nobody's ever shown it washes off all salmonella.'
Still, equally, nobody's shown it doesn't, and washing fruits and vegetables helps to prevent other illnesses too.
The Food Standards Agency says we should 'wash them under a running tap, or in a bowl of fresh water, making sure to rub their skin under the water'.
Begin with the least soiled produce first and give fruit and veg a last rinse after the scrub.
What are the symptoms of Salmonella?
Per the NHS, symptoms can take anywhere from six hours to three days (or more) after infection to show.
These include:
Loose stools (usually without blood or mucus)
Abdominal (tummy) pain
Fever, headache
Nausea (feeling sick) and/or vomiting
General tiredness.
Most people will recover in three to seven days. Many cases will go away on their own, but if you have severe symptoms, see your GP.
'In most cases, it is generally advised to wait until 48 hours after last having diarrhoea or sickness before returning to work or school/nursery,' the health service adds.
Washing hands, chilling and cooking food properly, and avoiding all 'raw' dairy can help prevent the disease.
Related...
RSD May Be 'Common' With ADHD – Here Are Its Symptoms
A New COVID Variant Is Here, And It's More Transmissible – Here Are The Signs And Symptoms
Dani Harmer's Not Alone – 62 Perimenopause Symptoms That Can Start In Your 30s
Orange background

Try Our AI Features

Explore what Daily8 AI can do for you:

Comments

No comments yet...

Related Articles

Dietary Emulsifiers and Their Role in IBD
Dietary Emulsifiers and Their Role in IBD

Medscape

time28 minutes ago

  • Medscape

Dietary Emulsifiers and Their Role in IBD

WASHINGTON, DC — A low-emulsifier-containing diet led to a threefold increased likelihood of improvement in symptoms of Crohn's disease compared with an emulsifier-containing diet in a randomized double-blind dietary trial involving 154 patients with mildly active disease living across the United Kingdom. The findings were reported at Gut Microbiota for Health (GMFH) World Summit 2025 by Benoit Chassaing, PhD, of the Institut Pasteur, Paris, France, whose research leading up to the trial has demonstrated that food additive emulsifiers —ubiquitous in processed foods — alter microbiota composition and lead to microbiota encroachment into the mucus layer of the gut and subsequent chronic gut inflammation. Patients in the ADDapt trial, which was also reported in an abstract earlier this year at the European Crohn's and Colitis Organization (ECCO) 2025 Congress, had a Crohn's disease activity index (CDAI) of 150-250 and evidence of inflammation (faecal calprotectin (FCP) ≥ 150 µg/g or endoscopy/radiology). All 'had been exposed in their regular diets to emulsifiers,' said Chassaing, a co-investigator, during a GMFH session on 'Dietary Drivers of Health and Disease.' They were randomized to either a low-emulsifier diet or to a low-emulsifier diet followed by emulsifier 'resupplementation' — a design meant to 'account for the very strong placebo effect that is always observed with dietary studies,' he said. All patients received dietary counseling, a smart phone app and barcode scan to support shopping, and weekly support. They also received supermarket foods for 25% of their needs that were either free of emulsifiers or contained emulsifiers, and they were provided three snacks per day that were emulsifier-free or contained carrageenan, carboxymethycellulse (CMC), and polysorbate-80 (P80) — dietary emulsifiers that are commonly added to processed foods to enhance texture and extend shelf-life. In the intention-to-treat (ITT) analysis, 49% of patients in the intervention group reached the primary endpoint of a 70-point reduction or more in CDAI response after 8 weeks compared with 31% of those in the control group ( P = .019), with an adjusted relative risk of response of 3.1 ( P = .003), Chassaing shared at the GMFH meeting, convened by the American Gastroenterological Association and the European Society of Neurogastroenterology and Motility. In the per-protocol analysis (n = 119), 61% and 47% of patients in the intervention and control groups, respectively, reached the primary outcome of CDAI response, with an adjusted relative risk of response of 3.0 ( P = .018), he said. Secondary endpoints included CDAI remission at 24 weeks, and according to the abstract for the ECCO Congress, in the ITT analysis, patients in the intervention group were more than twice as likely to experience remission. Chassaing noted at the GMFH meeting that as part of the study, he and coinvestigators have been investigating the participants' gut microbiota with metagenomic analyses. The study was led by Kevin Whelan, head of the Department of Nutritional Sciences at King's College London, London, England. Can Emulsifier-Sensitive Individuals Be Identified? In murine model research 10 years ago, Chassaing showed that the administration of CMC and P80 results in microbiota encroachment into the mucus layer of the gut, alterations in microbiota composition — including an increase in bacteria that produce pro-inflammatory flagellin — and development of chronic inflammation. Wild-type mice treated with these compounds developed metabolic disease, and mice that were modified to be predisposed to colitis had a higher incidence of robust colitis. Moreover, fecal transplantation from emulsifier-treated mice to germ-free mice reproduced these changes, 'clearly suggesting that the microbiome itself is sufficient to drive chronic inflammation,' he said. In recent years, in humans, analyses from the large French NutriNet-Sante prospective cohort study have shown associations between exposure to food additive emulsifiers and the risk for cardiovascular disease, the risk for cancer (overall, breast, and prostate), and the risk for type 2 diabetes. But to explore causality and better understand the mechanisms of emulsifier-driven changes on the microbiota, Chassaing and his colleagues also launched the FRESH study (Functional Research on Emulsifier in Humans), a double-blind randomized controlled-feeding study of the emulsifier CMC. For 11 days, nine healthy patients consumed an emulsifier-free diet and 11 consumed an identical diet enriched with 15 g/d of CMC. Patients on the CMC-containing diet had reduced microbiota diversity and depletions of an array of microbiota-related metabolites, but only a small subset had profound alterations in microbiota composition and increased microbiota encroachment into the mucus layer. 'Some seemed to be resistant to CMC-induced microbiota encroachment, while some were highly susceptible,' Chassaing said. The pilot study raised the question, he said, of whether there is an 'infectivity component' — some kind of 'sensitive' gut microbiota composition — that may be associated with dietary emulsifier-driven inflammation and disease. In other murine research, Chassaing and his team found that germ-free mice colonized with Crohn's disease-associated adherent-invasive E coli (AIEC) and subsequently given CMC or P80 developed chronic inflammation and metabolic dysregulation, 'clearly demonstrating that you can convert resistant mice to sensitive mice just by adding one bacteria to the ecosystem,' he said. 'The presence of AIEC alone was sufficient to drive the detrimental effects of dietary emulsifiers.' (In vitro research with transcriptomic analysis then showed that the emulsifiers directly elicit AIEC virulence gene expression, Chassaing and his coauthors wrote in their 2020 paper, facilitating AIEC's 'penetration of the mucus layer and adherence to epithelial cells and resulting in activation of host pro-inflammatory signaling.') 'We don't think it's solely the AIEC bacteria that will drive emulsifier sensitivity, though…we think it's more complex,' Chassaing said at the meeting. Overall, the findings raise the question of whether emulsifier-sensitive individuals can be identified. This, he said, is one of his most recent research questions. His lab has led the development of an in vitro microbiota model built to predict an individual's sensitivity to emulsifiers. In a study published in April, the model recapitulated the differential CMC sensitivity observed in the earlier FRESH study, suggesting that an individual's sensitivity to emulsifiers can indeed be predicted by examining their baseline microbiota. Interpreting the Epidemiology Chassaing's research arch illustrates the synergy between epidemiological research, basic/translational research, and clinical interventional research that's needed to understand the diet-microbiome intersection in inflammatory bowel disease, said Ashwin Ananthakrishnan, MBBS, MPH, associate professor of medicine at Massachusetts General Hospital, Boston, in an interview at the meeting. 'It's a good example of how to really span the spectrum, starting from the big picture and going deeper to understand mechanisms, and starting from mechanisms and expanding it out,' Ananthakrishnan said. In his own talk about research on IBD, Ananthakrishnan said that epidemiological data have shown over the past 10-15 years that total dietary fiber is inversely associated with the risk for Crohn's disease (with the strongest associations with fiber from fruits and vegetables). Studies have also shown that a higher intake of polyunsaturated fatty acids is associated with a lower risk for ulcerative colitis, whereas 'an n-6-fatty acid-rich diet is associated with a higher risk of ulcerative colitis,' he said. Dietary cohort studies, meanwhile, have shed light on the influence of dietary patterns — such as the Mediterranean diet and diets with high inflammatory potential—on IBD. A diet rich in ultra-processed foods has also been shown in a prospective cohort study to be associated with a higher risk for Crohn's disease, with certain categories of ultra-processed foods (eg, breads and breakfast foods) having the strongest associations. Such studies are limited in part, however, by inadequate assessment of potentially relevant variables such as emulsifiers, preservatives, and how the food is processed, he said. And in interpreting the epidemiological research on fiber and IBD, for instance, one must appreciate that 'there are a number of mechanisms by which fiber is impactful…there's a big picture to look at,' Ananthakrishnan said. Fiber 'can affect the microbiome, clearly, it can affect the gut barrier, and it can affect bile acids, and there are detailed translational studies in support of each of these.' But there are other constituents of fruits and vegetables 'that could potentially influence disease risk, such as AhR ligands and polyphenols,' he said. 'And importantly, people not eating a lot of fiber may be eating a lot of ultra-processed foods.' Most interventional studies of fiber have not shown a benefit of a high-fiber diet, Ananthakrishnan said, but there are multiple possible reasons and factors at play, including potential population differences (eg, in inflammatory status or baseline microbiota), shortcomings of the interventions, and potentially inaccurate outcomes. Abigail Johnson, PhD, RDN, associate director of the Nutrition Coordinating Center, University of Minnesota Twin Cities, which supports dietary analysis, said during the session that the focus of dietary research is 'moving toward understanding overall dietary patterns' as opposed to focusing more narrowly on vitamins, minerals, and macronutrients such as proteins, fats, and carbohydrates. This is an improvement, though 'we still don't have good approaches for understanding [the contributions of] things like additives and emulsifiers, food preparation and cooking, and food processing,' said Johnson, assistant professor in the Division of Epidemiology and Community Health at University of Minnesota Twin Cities. 'Perhaps by looking at things at the food level we can overcome some of these limitations.'

How to recognize - and overcome
How to recognize - and overcome

Yahoo

timean hour ago

  • Yahoo

How to recognize - and overcome

You're inferior. Incompetent. Unattractive. Everyone else is better than you are. If there are moments in your life when this is how you feel, it wouldn't be unusual. For some people, however, self-doubt colours all of their thinking and actions. They believe they're "good for nothing," "worthless" or "unloved." This can be an indication of an inferiority complex. "'Inferiority complex' isn't a medical term, but rather an everyday one," says Dr Gregor Müller, an instructor at the Institute of Psychological Psychotherapy (IPP) in Bochum, Germany. As he explains it, an inferiority complex is "a fundamental lack of self-assurance." You have no confidence in yourself, constantly see yourself as a failure and think you never do anything right. Psychological psychotherapist Eva Maria Klein prefers the term "self-esteem issues" to "inferiority complex." For people who have them, there's a discrepancy between their "ideal self and reality," she says. It arises when they place excessively high demands on themselves, often because of high performance expectations from their parents during childhood. If reality is at odds with what they come to expect of themselves, they suffer. Part of the problem, she says, is that people who expect and demand a lot of themselves continually compare themselves with others. While such comparisons aren't unusual, "drawing them constantly, and then feeling inferior, can weigh heavily on them." Feeling you don't measure up is often accompanied by further negative feelings. "Low self-esteem can also manifest itself in a fear of facing certain situations" and sometimes in a feeling of shame as well, which can make matters worse, Klein says. Depression can result. To determine whether you have an inferiority complex, simply consider your self-image. What knowledge and skills do you have? What are your strengths, your physical attributes? If you don't see much that's positive, but weaknesses and deficits instead, you might have a problem with your self-perception. "The more negatively you see yourself, and the stronger you believe that others see you negatively too, the more likely it is that you have an inferiority complex," Müller says. Feelings of inferiority and the negative thoughts underlying them can often be overcome. "In milder cases, even positive encouragement from friends or some positive feedback at work can be productive," says Müller. Apart from this, you can - and should - take steps yourself to pull up your low self-esteem. "Although the process may take a while, it can be successful," Klein says. There are several ways to do this, beginning with self-acceptance. "A first step can be accepting yourself as you are," leaving aside whether your self-perception is positive or negative, Müller says. In the second step, examine what it is you don't like about yourself, "for instance that you think you're unkempt or too fat." Then you make a plan to address the problems, such as washing your hair more often and always wearing clean clothes, or losing five to 10 kilograms in a certain amount of time. "Simply raising your awareness about a problem and planning to solve it can begin to boost your self-esteem," Müller says, since you've abandoned your passivity and taken action. "It can help to take your strengths and successes into account," Klein says. If you give it some thought, you'll surely come up with things about yourself that others find positive too. You can make a note of them, regularly add to the list, and - in phases of self-doubt - go over it to pick up your self-esteem. Know your own worth. Inferior? Worthless? People are never that, which is something you need to realize. Everyone has strengths and weaknesses. You can compare yourself with others, of course, and conclude there are certain things you don't do as well. "But that doesn't mean you're of less worth," Müller says. And perhaps you can do things that others can't. In some cases it can be difficult to overcome self-esteem issues on your own, particularly if they're linked with other symptoms and adversely affect your life. "Psychotherapy can then be effective," Klein says. Solve the daily Crossword

AI-Generated Medical Podcasts Deceive Even the Experts
AI-Generated Medical Podcasts Deceive Even the Experts

Medscape

timean hour ago

  • Medscape

AI-Generated Medical Podcasts Deceive Even the Experts

For the first time, researchers have evaluated the use of artificial intelligence (AI) to generate podcasts from peer-reviewed scientific articles. Using Google's NotebookLM application, the team created podcast scripts based on studies published in the European Journal of Cardiovascular Nursing ( EJCN ). The results were eye-opening: Half of the authors did not realize the podcast hosts were not human. The study assessed whether AI could simulate a realistic scientific dialogue between two speakers discussing published research. Findings were presented at this year's Annual Congress of the Association of Cardiovascular Nursing and Allied Professions and simultaneously published in EJCN . Too Polished to Be Human? The AI-generated podcasts averaged 10 minutes. Without knowing the content was machine-produced, most authors said their research was summarized clearly, in simple language, and with structured delivery. Some even remarked that the 'hosts' sounded like they had clinical or nursing backgrounds. But not all feedback was glowing. Several participants felt the delivery was unnaturally smooth — lacking hesitation, repetition, or organic back-and-forth — prompting suspicion of AI involvement. Others flagged mispronounced medical terms and factual errors. One podcast, for example, focused on heart failure diagnosis instead of management. Another spoke exclusively about women, even though the study included men. Some authors were also distracted by the overly enthusiastic, American-style tone of the narration, with superlatives used to describe modest results. A more academic tone, they suggested, would be more appropriate — particularly if the tool is used for scientific audiences. Promise for Science Communication Led by Philip Moons, PhD, from the KU Leuven Department of Public Health and Primary Care, Leuven, Belgium, the researchers created 10 podcasts based on EJCN articles. Despite imperfections, they concluded that 'AI-generated podcasts are able to summarize key findings in an easily understandable and engaging manner.' 'Podcasts were found to be most appropriate for patients and the public but could be useful for researchers and healthcare professionals as well if they were tailored accordingly,' the authors wrote. 'It was striking how accurate the podcasts were in general. Knowing that we are just at the beginning of this kind of AI-generated podcasts, the quality will become better over time, probably within the next few months,' Moons said in a press release. He believes the tool could help researchers more effectively disseminate their work. Moons got the idea after testing NotebookLM with one of his own papers, shortly after Google launched the feature in September 2024. 'When I did a first test case with one of my own articles, I was flabbergasted by the high quality and how natural it sounded.' After generating the podcasts — ranging from 5 to 17 minutes — researchers were asked to evaluate the content through a questionnaire and a 30-minute video interview. Missing Context but Strong Engagement All participating authors agreed that the podcasts effectively conveyed the key findings of their research in simple, accessible language. Many also found the conversational format between two 'hosts' made the content more engaging. Several praised the hosts' professionalism. 'I was curious about their background — it really seemed like they had medical or nursing training,' one author said. However, some were unsettled by the lack of introductory context. The podcasts provided no information about the identity of the speakers or how the audio was produced, leaving listeners uncertain about the source. Overall, most found the content reliable, though a few pointed out factual errors. One author noted that obesity was described as a 'habit,' potentially misleading listeners by implying it is merely a lifestyle choice. Despite these issues, half of the authors — one of whom was an AI expert — did not realize the podcasts were machine-generated. Many said they were 'shocked' or 'amazed' by the quality. Most of the participants were regular podcast listeners. Even those who suspected AI involvement were surprised by how natural and fluent the results sounded. Expanding Research Reach All authors agreed that future versions should clearly disclose AI involvement. Most also recommended adopting a more academic tone if the target audience includes researchers, along with a greater focus on study methods and limitations. Although patients and the general public were identified as the primary audience, the researchers noted that AI-generated podcasts could serve as a cost-effective, scalable way for healthcare professionals to stay current with new research. They also suggested the format could help broaden the visibility and reach of scientific publications. 'This could be a sustainable model to get the message out to people who do not typically read scientific journals,' Moons said. Still, he emphasized the need for human oversight 'to add nuance.' He envisions a hybrid model in which AI-generated content is supplemented with human input. That vision may already be taking shape. The beta version of Google's NotebookLM (currently available only in English) now allows real-time interaction with the AI. After launching a podcast, users can ask questions directly to one of the 'hosts.' The AI generates a spoken response, and the podcast then continues — seamlessly integrating human-machine dialogue.

DOWNLOAD THE APP

Get Started Now: Download the App

Ready to dive into a world of global content with local flavor? Download Daily8 app today from your preferred app store and start exploring.
app-storeplay-store